In addition to TFs, amphipathic helix proteins, SNL1 and SNL2, were recently shown to form another cross-link point of ABA and ethylene signalling . They belong to SWI-INDEPENDENT3 (SIN3)-LIKE protein family, which play roles in recruiting histone binding proteins resulting in a transcriptionally repressed state of the chromatin . During Arabidopsis seed dormancy establishment, SNL1 and SNL2, through histone deacetylation, negatively regulates the ethylene pathway while positively regulates the ABA pathway, leading to an increase in seed dormancy. These two proteins regulate key components of ethylene ( ACO 1, ACO 4, ERF9 and ERF1 12) and ABA ( CYP707A1 & A2 , and NCED4 ) pathway by modifying their histone acetylation levels, thus promoting the seed dormancy . Interestingly, only NCED4 gene expression was down regulated in the double mutant snl1 snl2 (low ABA levels) raising the question whether increased ABA levels would induce SNLs proteins that in turn could repress ethylene signalling is unknown, which deserves further studies.
There is also a study of the brassinosteroid, 28-Homobrassinolide (HB), in rats which can also be used as a proxy for Laxogenin. The study concluded that HB stimulated protein synthesis and inhibited protein degradation in part by inducing Akt phosphorylation .  Akt is a serine/threonine kinase that signals downstream of growth factor receptors and phosphoinositide-3 kinase PI3K. Akt also stimulates glucose uptake, glycogen synthesis via Akt/mTOR and Akt/GSK-3β signaling networks.  In addition, HB triggered a selective anabolic response with minimal to no androgenic side-effects.